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|Title:||Zeeman-type dragging in the Kerr-Newman and Nut spacetimes|
|Abstract:||Traditional cameras have a narrow field of view, to enlarge the field of view omnidirectional cameras can be used. In this work, we propose, a simple an elegant solution to the image formation model for omnidirectional cameras with parabolic mirrors. We propose the use of conformal geometric algebra (CGA), since the involved transformation operations in the model can be represented as an special group of multivectors. This representation is advantageous since the inversions are linearized, furthermore the transformation can be applied to all the geometric objects of the CGA. In consequence, the paracatadioptric image formation can be simplified, since the procedure is the same for points, point-pairs, lines, or circles. As an application example the control of a nonholonomic mobile robot using paracatadioptric line images and the proposed framework is described. " 2010 IEEE.",,,,,,"10.1109/ROBOT.2010.5509507",,,"http://hdl.handle.net/20.500.12104/45689","http://www.scopus.com/inward/record.url?eid=2-s2.0-77955820942&partnerID=40&md5=149251c0bc653b5083af9f64ea7ce8ff",,,,,,,,"Proceedings - IEEE International Conference on Robotics and Automation",,"2543|
2548",,,,"Scopus",,,,,,,,,,,,"Vision-based robot control with omnidirectional cameras and conformal geometric algebra",,"Conference Paper" "47477","123456789/35008",,"Correa-Cerro, L., Department of Medical Genetics, University of Ulm, Albert-Einstein-Allee 11, D-89069 Ulm, Germany, Doctorado en Genética Humana, University of Guadalajara, México, Mexico; Berthon, P., Saint Louis Hospital, CeRePP-JE2153-University Paris VII, Paris, France; Haussler, J., Department of Medical Genetics, University of Ulm, Albert-Einstein-Allee 11, D-89069 Ulm, Germany; Bochum, S., Department of Medical Genetics, University of Ulm, Albert-Einstein-Allee 11, D-89069 Ulm, Germany; Drelon, E., Saint Louis Hospital, CeRePP-JE2153-University Paris VII, Paris, France; Mangin, P., Saint Louis Hospital, CeRePP-JE2153-University Paris VII, Paris, France; Fournier, G., Service d'Urologie, CHU de la Cavale Blanche, Brest, France; Paiss, T.; Cussenot, O., Saint Louis Hospital, CeRePP-JE2153-University Paris VII, Paris, France; Vogel, W., Department of Medical Genetics, University of Ulm, Albert-Einstein-Allee 11, D-89069 Ulm, Germany",,"Correa-Cerro, L.
Vogel, W.",,"1999",,"Polymorphisms in the vitamin D receptor (VDR) gene have been analyzed in several studies for an association with prostate cancer (PCA) and odds ratios (OR) ? 3 have been observed in study populations from North America. We studied three polymorphisms in the VDR gene (poly-A microsatellite, TaqI and FokI RFLPs) in 105 controls and 132 sporadic PCA cases from France and in a collection of families from Germany and France. The polymorphisms near the 3' end of the gene were in linkage disequilibrium with an almost complete coincidence of the short poly-A alleles and t (presence of the restriction site) of the TaqI polymorphism, (contingency tables, P < 0.0001). An association was found by logistic regression for the poly-A between PCA and the heterozygous genotype (S/L; S < 17, L ? 17, OR = 0.44, 95% confidence interval, CI = 0.198-0.966, P = 0.041). OR was lower in patients ? 70 years old and patients with a Gleason score ? 6. The Tt genotype of the TaqI RFLP also showed an association with PCA (OR = 0.5, CI = 0.27-0.92, P = 0.026). This association was also stronger for patients ? 70 years old (OR = 0.31, CI = 0.15-0.63, P = 0.001). The risk alleles were S and t alleles as indicated by the OR of the homozygotes, although these were not significant. The FokI RFLP at the 5' end of the gene did not reveal any association (P > 0.7). While some association studies differ between Europe and North America, our present findings with the VDR gene agree with those from North America, indicating a weak but general role of the VDR in PCA susceptibility.",,,,,,"10.1007/s004390051102",,,"http://hdl.handle.net/20.500.12104/45698","http://www.scopus.com/inward/record.url?eid=2-s2.0-0032843269&partnerID=40&md5=163af3bb6acbe39472e63bc3562315d2",,,,,,"3",,"Human Genetics",,"281
WOS",,,,,,,,,,,,"Vitamin D receptor polymorphisms as markers in prostate cancer",,"Article" "47570","123456789/35008","Rivera-Cervantes, M., Departamento de Biologia Celular y Molecular, Centro Universitario de Ciencias Biologicas y Agropecuarias, Universidad de Guadalajara, Km. 14.5 Carretera A Nogales, Predio Las Agujas Nextipac, Zapopan, Jalisco, Mexico; Feria-Velasco, A.I., Departamento de Biologia Celular y Molecular, Centro Universitario de Ciencias Biologicas y Agropecuarias, Universidad de Guadalajara, Km. 15.5 Carretera-Guadalajara-Nogales, Predio Las Agujas, Zapopan, Jalisco, Mexico; Junyent, F., Unitat de Bioquimica, Facultat de Medicina i Ciencies de la Salut, Universitat Rovira i Virgili, C./ St. Llorenc 21 Reus (Tarragona), Barcelona, Spain; Espuny, A.C., Department of Pharmacology and Biomedical Chemistry, University of Barcelona, Avenida Diagonal 643, Barcelona, Spain; Beas-Zárate, C., Departamento de Biologia Celular y Molecular, Centro Universitario de Ciencias Biologicas y Agropecuarias, Universidad de Guadalajara, Km. 15.5 Carretera-Guadalajara-Nogales, Predio Las Agujas, Zapopan, Jalisco, Mexico
Beas-Zárate, Carlos., Universidad de Guadalajara. Centro Universitario de Ciencias Biológicas y Agropecuarias",,"Rivera-Cervantes M
Beas-Zárate, Carlos",,"2013",,"Epilepsy has been characterized a disease whose social and occupational behavioural has had devastating economical consequences and is associated with great cumulative brain damage and neurological deficits. From different forms of epilepsy, the most frequent type is temporal lobe epilepsy (TLE), being the most common form of drug refractory epilepsy. Although there are a great amount of studies about the mechanisms involved in neuronal damage and death during critical phases of epileptogenesis, it is crucial to construct strategies for neuroprotection that may prevent the development of epilepsy. In this chapter, some molecular mechanisms involved in the neuronal death, which are induced by excitotoxicity phenomena following the signalling pathways activation and studied in animal models under seizure conditions or expressed in the epilepsy are discussed, mainly those as the mitogen-activated protein kinases, Jak/Stat, and Pi3k/Akt pathways those genes responsible to participate in the apoptosis and cell cycle regulation are also analysed. In summary, the structural and molecular changes at cellular level are believed to play a key role in the generation of convulsive seizures and its possible identification should facilitate the develop of potential therapeutic targets heading towards of specific genes, proteins, and signalling pathways altered during the different stages of epileptogenesis process. © 2013 Springer Science+Business Media, LLC. All rights are reserved.",,,,,,"10.1007/978-1-4614-6464-8_6",,,,"http://www.scopus.com/inward/record.url?eid=2-s2.0-84929555997&partnerID=40&md5=8fe824ad62289b4a869dccc5ca84eca8
http://hdl.handle.net/20.500.12104/45791",,,,,,,,"Pharmacoresistance in Epilepsy: From Genes and Molecules to Promising Therapies",,"77
97",,,,"Scopus",,,,,,"Apoptosis; Cell damage; Cell death; Epileptogenesis; Excitotoxicity; Extrinsic pathway; Intrinsic pathway; TLE",,,,"Intracellular pathways associated with neuronal survival and death in epilepsy",,,,"Book Chapter", "47554","123456789/35008",,"Mitskievich, N.V., Department of Physics, CUCEI, Universidad de Guadalajara, Apartado Postal 1-2011, C.P. 44100, Guadalajara, Jalisco, Mexico; Benitez, L.I.L., Mathematics and Physics Department, Instituto Tecnológico de Estudios Superiores de Occidente A.C., Periferico Sur Manuel Gomez Marín 8585, Tlaquepaque, Jal., C.P. 45090, Mexico",,"Mitskievich, N.V.
Benitez, L.I.L.",,"2008",,"In this communication we discuss two distinct Zeeman-type gravitomagnetic effects deserving attention since they can be easily characterized in their exact form, not via approximation procedures. Some observations are also made on gravitoelectric effects. " 2008 World Scientific Publishing Co. Pte. Ltd.
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