Please use this identifier to cite or link to this item: https://hdl.handle.net/20.500.12104/42408
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dc.contributor.authorRivera, H.
dc.contributor.authorDiaz-Castanos, L.
dc.date.accessioned2015-09-15T18:15:55Z-
dc.date.available2015-09-15T18:15:55Z-
dc.date.issued1992
dc.identifier.urihttp://www.scopus.com/inward/record.url?eid=2-s2.0-0027058256&partnerID=40&md5=3c7faad60d3e1f6b94a1f52429498655
dc.identifier.urihttp://hdl.handle.net/20.500.12104/42408-
dc.description.abstractBalanced Y;Autosome translocations seem to be associated with male infertility whenever the Y breakpoint is located into Yq11 (where the azoospermia factor maps) or near its boundaries. By analogy with the effect on female carriers' fertility of balanced X;Autosome translocations, the band Yq11 emerges as the critical segment. This hypothesis implies that the subjacent mechanism is an impaired expression of the azoospermia factor - either through its physical disruption or via a position effect - rather than a disorder of the sex bivalent inactivation during prophase I.
dc.relation.isreferencedbyScopus
dc.titleIs Yq11 the main critical segment in balance Y;Autosome translocations?
dc.typeArticle
dc.relation.ispartofjournalAnnales de Genetique
dc.relation.ispartofvolume35
dc.relation.ispartofissue4
dc.relation.ispartofpage224
dc.relation.ispartofpage226
dc.subject.keywordazoospermia factor; critical segment; male sterility; Y;Autosome translocations
dc.contributor.affiliationRivera, H., Division de Genetica, Unidad de Investigacion Biomedica, Centro Medico de Occidente, Apartado Postal 1-3838, Guadalajara, Mexico; Diaz-Castanos, L., Division de Genetica, Unidad de Investigacion Biomedica, Centro Medico de Occidente, Apartado Postal 1-3838, Guadalajara, Mexico
Appears in Collections:Producción científica UdeG

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